Diabetes

Diabetes like any disease comes about as a malfunction of one or more of your bodies systems.  Much if not most of the time this is a result of a slow degenerative process due to the lack of adequate bodily supplies of the elements necessary for normal function and rejuvenation of affected organs. Commercial Farming and natural erosion has depleted global farmlands of most essential elements therefore it is not wise to assume that your diet contains enough of these elements for normal body function and maintenance. See Senate Document 264. Does what you eat provide all the essential elements you need? See Senate Document 264. Just how good is the "best" diet?  See The Food Pyramid

  DIABETES is a chronic metabolic disorder that adversely affects the body's ability to manufacture and use insulin, a hormone necessary for the conversion of food into energy. The disease greatly increases the risk of blindness, heart disease, kidney failure, neurological disease and other conditions for the approximately 16 million Americans who are affected by it. Type I, or juvenile onset diabetes, is the more severe form of the illness.
  Type I diabetes is what is known as a 'complex trait', which means that mutations in several genes likely contribute to the disease. For example, it is now known that the insulin-dependent diabetes mellitus (IDDM1) locus on chromosome 6 may harbor at least one susceptibility gene for Type I diabetes. Exactly how a mutation at this locus adds to patient risk is not clear, although a gene maps to the region of chromosome 6 that also has genes for antigens (the molecules that normally tell the immune system not to attack itself). In Type I diabetes, the body's immune system mounts an immunological assault on its own insulin and the pancreatic cells that manufacture it. However, the mechanism of how this happens is not yet understood.
  About 10 loci in the human genome have now been found that seem to confer susceptibility to Type I diabetes. Among these are (1) a gene at the locus IDDM2 on chromosome 11 and (2) the gene for glucokinase (GCK), an enzyme that is key to glucose metabolism which helps modulate insulin secretion, on chromosome 7.
  Conscientious patient care and daily insulin dosages can keep patients comparatively healthy. But in order to prevent the immunoresponses that often cause diabetes, we will need to experiment further with mouse models of the disease, and advance our understanding of how genes on other chromosomes might add to a patient's risk of diabetes.